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Saturday, August 1, 2020 | History

4 edition of CCK, dietary variety, and food intake in rats found in the catalog.

CCK, dietary variety, and food intake in rats

by Tammy L. B. McKenzie

  • 113 Want to read
  • 37 Currently reading

Published by Brock University, Dept. of Psychology in St. Catharines, Ont .
Written in English

    Subjects:
  • Ingestion -- Regulation.,
  • Ingestion -- Psychological aspects.,
  • Cholecystokinin.,
  • Rats.

  • Edition Notes

    Statementby Tammy L.B. McKenzie.
    The Physical Object
    Paginationxv, 150 leaves :
    Number of Pages150
    ID Numbers
    Open LibraryOL23340453M

    1. Introduction. Hypothalamic orexin neurons have been implicated in the control of food intake in many studies (reviewed in [1,2,48]).These cells express a prepro-gene that is post-translationally modified to yield the isoforms orexin-A and orexin-B. Orexin-A, which binds to the orexin 1 and orexin 2 receptors (OX1R; OX2R), affects food intake when administered into the lateral, 3rd or 4th.   There is a physiological decline in food intake with aging. The reasons for the decline in food intake are multifactorial and involve both peripheral and central mechanisms. Altered hedonic qualities of food occur due to alterations in taste and, more particularly, smell with aging.

    Rats that lack the CCK-A receptor become obese, but transgenic mice lacking CCK-A receptors do not become obese. CCK inhibits food intake in human subjects. Enterostatin, the pentapeptide produced when pancreatic colipase is cleaved in the gut, has been shown to reduce food intake. This peptide differs in its action from CCK by selectively. Abstract. The control of food intake consists of neural and hormonal signals between the gut and central nervous system (CNS). Gut hormones such as CCK, PYY and PP signal to important areas in the CNS involved in appetite regulation to terminate a meal.

    CCK is a prototypic satiation peptide that is secreted by the duodenal and jejunal mucosa primarily in response to fat and protein ingestion, and it decreases food intake rapidly but transiently via the activation of vagal afferents (12, 19, 20). CLASSIC Dietary Fat Content and Caloric Intake. Lauren Lissner, David A. Levitsky, Barbara J. Strupp, Heidi J. Kalkwaf, Daphne A. Roe. Dietary fat and the regulation of energy intake in human subjects. The American Journal of Clinical Nutrition .


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CCK, dietary variety, and food intake in rats by Tammy L. B. McKenzie Download PDF EPUB FB2

To explore the effect of CCK on food intake in Siberian sturgeon, cck cDNA sequence of bp was obtained, and cck mRNA possessed the highest expression in brain. The expressions of cck Cited by: 8. Request PDF | CCK, dietary variety, and food intake in rats / | Among the environmental factors that can affect food intake is the extent of dietary variety available in the environment.

Numerous Author: Tammy Mckenzie. CCK, dietary variety, and food intake in rats / By Tammy L. McKenzie. Abstract. Among the environmental factors that can affect food intake is the extent of dietary variety available in the environment.

Numerous studies have demonstrated that variety in a meal can increase the amount of food consumed in humans, rats, and other : Tammy L.

McKenzie. Other studies have corroborated the reduction of food intake by trypsin inhibitors, attributing this effect to CCK induction [15,18,20,21,22]. Komarnytsky et al. reported that oral administration of a concentrated potato trypsin inhibitor (CIPP) was efficient in reducing food intake and weight gain in eutrophic rats.

This was due to increased Cited by: Cholecystokinin Decreases Food Intake in Rats 1 Article (PDF Available) in Journal of Comparative and Physiological Psychology 84(3) October with Reads How we measure 'reads'. CCK-1 receptors influence satiety, as CCK-1 agonists reduce food int11 whereas pharmacological blockage of CCK-1 receptors stimulate food int12,13,14,15,16,17,18,   The hormone CCK, originally purified inexerts a wide variety of effects on the low-carbohydrate diet exhibit reduced satiety in response to CCK injection and reduce their food intake less in response to CCK than rats consuming a high it was demonstrated that rats prone to dietary-induced obesity exhibit.

In this experiment we controlled for this possibility, by comparing the response to CCK ( μg/kg) in rats adapted to LF diet and tested for reduction of food intake by CCK on low fat (LF/LF, n = 6) with the response of rats adapted to low fat, but tested for CCK satiety on a high-fat diet.

After a 17 hr fast, rats were given intraperitoneal injections of saline or ascending doses of to μg/kg of the synthetic octapeptide of CCK.

On all diets, obese rats required higher doses of CCK to significantly reduce feeding and showed smaller intake reductions than lean rats (p. A significant reduction in food intake was seen in the first (%) and second (%) hour following oral administration of mg kg −1 PPIC to 6 h fasted rats in the early dark phase (P.

Hence, unsulfated CCK and gastrin do not significantly reduce food intake. Reductions of food intake following injection of exogenous CCK, or release of endogenous CCK after intestinal nutrient infusions, are attenuated or abolished by CCK-1 receptor antagonists in the human and the rat, while systemic administration of CCKreceptor.

Acute and prolonged effects of oral PPIC administration ( mg/kg per day) on food intake, body weight, and gastric emptying were evaluated in healthy rats. Parameters of body weight, food intake, plasma glucose, insulin, and cholecystokinin (CCK) were measured. Duodenal proteolytic activity and CCK expression were determined in tissue extracts.

Dietary fat has frequently been blamed for the increase in prevalence of obesity (Bray et al., ). Epidemiological studies have demonstrated a positive relationship between high-fat diets and excess energy intake due to their high energy density and palatability (Prentice and Poppitt, ).

However, this association is confounded by differences in physical activity, smoking, and food. Rats that lack the CCK-A receptor become obese, but transgenic mice lacking CCK-A receptors do not become obese.

CCK inhibits food intake in human subjects. Enterostatin, the pentapeptide produced when pancreatic colipase is cleaved in the gut, has been shown to reduce food intake.

The effects of intracerebroventricular administration of devazepide, a CCK(1) receptor antagonist, was investigated on food intake in rats. In the first experiment, rats (n=5) were deprived of. 1. CCK and satiety. Gibbs et al. () originally demonstrated the ability of exogenously administered cholecystokinin (CCK) to inhibit food intake in rats.

Their characterization of the pattern of behavioural changes produced by CCK suggested that CCK administration resulted in. The effect of S, above the % level, on food intake was marked, immediate and dose dependent ().Mean total food intakes per group over the 8-week study were not different between the control.

CCK-induced c-fos protein expression in the NTS of gastrectomized rats 3 weeks and 3 months after surgery was also compared to weight-matched unoperated control rats, since body weight might influence the sensitivity to CCK with respect to food intake 21 (3 weeks after surgery: gastrectomy, n = 5, body weight ± 24 g; weight-matched control.

Extracts of kidney beans (Phaseolus vulgaris) are known to reduce food intake and glycemia in rodents and humans. This study evaluated the effect of a novel extract of P. vulgaris on food (regular food pellets, starch-enriched diet, and chocolate-flavored beverage) intake, body weight, and glycemia in rats.

The effect of the combination of the colecistokinin (CCK) receptor antagonist. Intravenous infusions of CCK-8 ( g/rat) produced reduction of sucrose intake in both capsaicin-and vehicle-treated rats at all three time points (P ), while g/rat failed to reduce.

Exendin-4 dose dependently decreased food intake to a greater extent in S5B rats compared to OM rats. The intake of low-fat diet, compared to the intake of high-fat diet, was more sensitive to the.The relationship of body weight status and fiber effect on energy intake suggests that obese individuals may be more likely to reduce food intake (Evans and MillerPorikos and Hagamen ) with dietary fiber inclusion.

However, many more studies will be required to establish this relationship firmly by comparing directly the effects of. If rats are allowed unlimited access to a high-energy palatable diet, their energy intake diverges quickly from control animals fed a bland diet, but the difference in energy intake stabilizes within days (Archer and Mercer, ).

Thus rats, which presumably do not feel societal pressure to be slim, will overeat palatable food, but only to a.